Vascular involvement in cranial hyperostosis.

Various skeletal dysplas ias known to produce c ranial hyperostosis include osteopetrosis, c rani ometaphyseal dysplas ia, hereditary hyperphosphatasia, Van Buchem disease, and Engelmann disease [1]. Neurologic compl ications such as optic atrophy, facial paralysis, or deafn ess may result from progressive encroachment on the cranial foramina due to bony overgrowth at the base of the skull . Narrowing and compromise of the arteri es and veins at the skull base by the same mechanism is not as well known or documented. This paper deals with the computed tomographic (CT) and angiographic findings in a child with hyperostosis of the skull base due to craniometaphyseal dysplasia.